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     <title><![CDATA[NUST Institutions Library Catalogue Search for 'kw,wrdl: (su-br:&quot;Behavioral assessment.&quot;)']]></title>
     <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-search.pl?idx=kw&amp;q=%28su-br%3A%22Behavioral%20assessment.%22%29&amp;format=rss</link>
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     <description><![CDATA[ Search results for 'kw,wrdl: (su-br:&quot;Behavioral assessment.&quot;)' at NUST Institutions Library Catalogue]]></description>
     <opensearch:totalResults>9</opensearch:totalResults>
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     <item>
       <title>
    Geriatric neuropsychology :


    assessment and intervention /





</title>
       <dc:identifier>ISBN:1593852266 (cloth : alk. paper)</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=2439</link>
        
       <description><![CDATA[









	   <p>
	   New York : Guilford Press, 2006
                        . xii, 467 p. :
                        
                         27 cm.. 
                         1593852266 (cloth : alk. paper)
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=2439">Place Hold on <em>Geriatric neuropsychology :</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=2439</guid>
     </item>
	 
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     <opensearch:Query role="request" searchTerms="" startPage="" />
     <item>
       <title>
    Urdu translation, adaptation and assessment of psychometric properties of brief COPE scale/






</title>
       <dc:identifier>ISBN:</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=342239</link>
        
       <description><![CDATA[









	   <p>
	   Islamabad S3H NUST 2017
                        . 78p
                        
                        
                        
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=342239">Place Hold on <em>Urdu translation, adaptation and assessment of psychometric properties of brief COPE scale/</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=342239</guid>
     </item>
	 
     <atom:link rel="search" type="application/opensearchdescription+xml" href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-search.pl?&amp;sort_by=&amp;format=opensearchdescription"/>
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     <item>
       <title>
    Characterization of atrial natriuretic peptide (ANP) response in depression-induced mice models /






</title>
       <dc:identifier>ISBN:</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=608667</link>
        
       <description><![CDATA[









	   <p>By Ali, Saman . 
	   
                        . 71p.
                        , Depression is a complex psychological disorder that is also often link to the hormonal
imbalances. Our research delves into the intricate relationship between depression and
atrial Natriuretic Peptide (ANP), considering the multifaceted influences of neurological,
genetic, and environmental factors. With a focus on hormonal imbalances as key
contributors to depression, our investigation explores the potential therapeutic effects of
Levothyroxine (LT4) in the context of ANP. Utilizing an early weaning mouse model
involving maternal separation, we conducted a detailed examination of the anxiolytic
effects of LT4, aiming to evaluate its efficacy in alleviating symptoms associated with
anxiety and depression. Behavioral assessments and histological analyses were employed
to comprehensively evaluate the impact of LT4 on ANP. This study also extends to
molecular investigations using RT-PCR to analyze the distribution and expression of ANP
within the mouse central nervous system, highlighting the cortex region. Our findings
reveal significant differences in brain expression levels of ANP between treated mice and
those exhibiting depressive symptoms. This insight suggests potential therapeutic
applications of ANP for mitigating depression, presenting intriguing avenues for further
research, particularly in the context of depression-induced mouse models through parental
separation. This research contributes to an enriched understanding of the complex factors
influencing depression and proposes interventions that extend beyond conventional
approaches. The integration of behavioral assessments, histological analyses, and
molecular investigations offers a holistic perspective, laying the groundwork for future
exploration in the critical realm of mental health research.
                         30cm. 
                        
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=608667">Place Hold on <em>Characterization of atrial natriuretic peptide (ANP) response in depression-induced mice models /</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=608667</guid>
     </item>
	 
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     <item>
       <title>
    Therapeutic Potential of Galantamine for Managing the Complications Associated with Ischemic Stroke /






</title>
       <dc:identifier>ISBN:</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=608668</link>
        
       <description><![CDATA[









	   <p>By Ahsan, Mehwish . 
	   
                        . 104p.
                        , Ischemic stroke remains the leading cause of illness and second leading cause of death
worldwide. Previous research has shown that galantamine has neuroprotective properties,
reducing neuronal death and damage in neurodegenerative conditions and improving cognitive
function in Alzheimer's disease patients. The investigation looked into the possible
mechanisms by which this medication could reduce cell death. Wistar rats were subjected to a
temporary 30-minute occlusion of the right middle cerebral artery (MCA) and given an oral
dose of 5mg/kg for three weeks. After 18 days of surgery, behavioral assessments were carried
out. Galantamine enhanced grip strength, motor function, and muscle strength in rats. Spatial
memory and object recognition examinations revealed cognitive improvements, thus indicating
enhanced cognitive abilities and memory retention. Moreover, rats subjected to galantamine
treatment displayed increased sociability and heightened locomotor activity during social
interaction and open-field assessments. Molecular analysis of galantamine treatment in rats
showed an upregulation of SOD2 expression, suggesting enhanced antioxidant defense, and a
downregulation of TLR4 expression, suggesting a reduction in neuroinflammation, supporting
the anti-inflammatory properties of galantamine. The histological analysis done with H&amp;E
staining of brain tissue revealed enhanced tissue morphology in the galantamine-treated group,
signifying the neuroprotective effects of galantamine. The reduction in neuronal damage,
edema, and inflammatory cell infiltration further supported this observation. The results
demonstrate that galantamine, potentially through the preservation of a functional cholinergic
system, mitigated the impairments caused by stroke in a basic learning and memory test by
decreasing cellular death.
                         30cm. 
                        
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=608668">Place Hold on <em>Therapeutic Potential of Galantamine for Managing the Complications Associated with Ischemic Stroke /</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=608668</guid>
     </item>
	 
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     <item>
       <title>
    Nelson essentials of pediatrics /






</title>
       <dc:identifier>ISBN:9780323775632  | 9780323775625</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=612290</link>
        
       <description><![CDATA[









	   <p>
	   
                        . xv, 829 pages;
                        
                         29 cm.. 
                         9780323775632  | 9780323775625
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=612290">Place Hold on <em>Nelson essentials of pediatrics /</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=612290</guid>
     </item>
	 
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     <opensearch:Query role="request" searchTerms="" startPage="" />
     <item>
       <title>
    Effect of Tactile Stimulation on Visual Memory Performance /






</title>
       <dc:identifier>ISBN:</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=612584</link>
        
       <description><![CDATA[









	   <p>By Latif, Asma . 
	   
                        . 80p.
                        , The human eye continuously perceives information about surroundings to be processed
and stored in memory so that it can be retrieved. Environmental factors may have
positive and negative effects on memory performance and human cognitive processing.
Many studies have addressed the effect of auditory circumstances on spatial tasks and
visual memory performance. However, only a few studies highlighted the cross-modal
interaction between vision for visual cue and touch for training of same visual pattern in
tactile pattern. In addition, very little research has been conducted on the effect of tactile
stimulation towards memorizing visual tasks. The main objective of this study is to
investigate the effect of visuo-tactile stimulation on adult memory. Sixty-two subjects
participated in this behavioral study having normal and corrected to normal vision.
Participants are divided into two groups and each subject goes thorough Mini mental
state examination and Edinburg handedness inventory. The visual assessment task
consists of different shapes along with three-digit numbers. During the memorization
period, visual assessment task was displayed on computer screen and tactile stimulation
was delivered on index finger of the dominant hand of the participant. The participant
was provided with an evaluation sheet containing shapes only. If the shape is paired with
its corresponding number, then it was be considered correct. The p-value &lt; 0.05 in visual
assessment test showed a significant effect of tactile stimulation on visual memory
performance. The findings of this study concluded that participants memorized the object
number pair task better in the presence of tactile stimulation as compared to control/no
stimulation. One of the conclusions of our work is that combing vision and touch sense
may improve cognitive ability and may be provided to people during learning and
remembering visual tasks. For future recommendations, heterogenous sample along with
brain response can be studied.
                         30cm. 
                        
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=612584">Place Hold on <em>Effect of Tactile Stimulation on Visual Memory Performance /</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=612584</guid>
     </item>
	 
     <atom:link rel="search" type="application/opensearchdescription+xml" href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-search.pl?&amp;sort_by=&amp;format=opensearchdescription"/>
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     <item>
       <title>
    Multi-Task Learning using Brain Computer Interface /






</title>
       <dc:identifier>ISBN:</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=614570</link>
        
       <description><![CDATA[









	   <p>By Fazal, Mariyam . 
	   
                        . 102p.
                        , Brain-computer interfaces (BCIs) can decode not only what users are thinking but also the intensity
of their cognitive effort. However, BCIs have traditionally been constrained to single-task
applications. Multitask learning (MTL) offers a promising solution by enabling BCIs to handle
multiple related tasks simultaneously, enhancing both performance and usability.This study applied
MTL to EEG data from N-back working memory tasks (0-back, 2-back, and 3-back) using openaccess data from 26 participants at Technische Universität Berlin. We developed a novel hybrid
CNN-LSTM-tAPEformer architecture that integrates Convolutional Neural Networks for spatial
feature extraction, Long Short-Term Memory networks for temporal sequence modeling, and
Transformer blocks with specialized attention mechanisms for capturing long-range temporal
dependencies. The proposed model performs dual functions by classifying accurate behavioral
responses while simultaneously measuring cognitive workload across varying task complexity
levels. Notable innovations include the development of Time Absolute Position Encoding (tAPE)
that enhances temporal processing by integrating sinusoidal positional encoding with adaptive
channel-specific encoding to preserve temporal relationships in EEG data. The system incorporates
regional and temporal self-attention mechanisms along with global attention pooling to achieve
enhanced neural pattern detection. Through leave-one-subject-out cross-validation methodology,
the model was trained using data from all participants except one, then evaluated on the excluded
individual to assess cross-subject generalization performance. Findings validate the hybrid CNNLSTM-tAPEformer model's efficacy for practical multi-task learning implementations,
establishing its utility for BCI applications that demand concurrent cognitive state identification
and workload assessment.
                         30cm. 
                        
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=614570">Place Hold on <em>Multi-Task Learning using Brain Computer Interface /</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=614570</guid>
     </item>
	 
     <atom:link rel="search" type="application/opensearchdescription+xml" href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-search.pl?&amp;sort_by=&amp;format=opensearchdescription"/>
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     <item>
       <title>
    Therapeutic Effects of Sound-Mediated Stimulation in a Mouse Model of Depression - A Comparative Study /






</title>
       <dc:identifier>ISBN:</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=615203</link>
        
       <description><![CDATA[









	   <p>By Reemaz, Tooba . 
	   
                        . 90p.
                        , Depression is a prevalent neuropsychiatric disorder characterized by mood disturbances, cognitive
impairment, and behavioral deficits. While pharmacological treatments such as Fluoxetine are
widely used, they often represent limitations including delayed onset of action and side effects,
highlighting the need for alternative, non-invasive therapeutic strategies. One promising approach
is neuromodulation through gamma entrainment using auditory stimulation, aimed at restoring
disrupted neuronal synchronization. This study evaluates the effects of 40 Hz sound stimulation
compared to fluoxetine in a chronic restraint stress-induced mouse model of depression,
hypothesizing that sound therapy will yield superior recovery outcomes. Mice were divided into
control, depressed, fluoxetine-treated, and sound stimulation-treated groups. Behavioral
assessments, including the Tail Suspension Test, Forced Swim Test, Social Interaction Test,
Sucrose Preference Test, Open Field Test, and Light-Dark Box Test, revealed that 40 Hz sound
therapy significantly alleviated depressive-like behaviors, often outperforming fluoxetine.
Histopathological analysis focused on the hippocampus and prefrontal cortex showed that sound
stimulation preventing neuronal loss and parvalbumin-expressing interneurons, which are crucial
for inhibitory circuit function and gamma oscillatory activity. Molecular evaluation through qPCR
for pavlb and brain-derived neurotrophic factor; BDNF supported these findings, displaying
increased expression levels indicating enhanced interneuron integrity and neuroplasticity. This
neuromodulatory treatment encourages the molecular and functional repair of brain circuitry
implicated in depression. The findings demonstrated 40 Hz sound entrainment's ability to change
brain oscillations and ameliorate cellular and behavioral deficiencies linked to depression,
indicating that it may be a novel supplement or substitute for traditional antidepressant treatments.
                         30cm. 
                        
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=615203">Place Hold on <em>Therapeutic Effects of Sound-Mediated Stimulation in a Mouse Model of Depression - A Comparative Study /</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=615203</guid>
     </item>
	 
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     <opensearch:Query role="request" searchTerms="" startPage="" />
     <item>
       <title>
    Social Media Analytics for Mental Health Assessment /






</title>
       <dc:identifier>ISBN:</dc:identifier>
        
        <link>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=615943</link>
        
       <description><![CDATA[









	   <p>By Liaquat, Umna . 
	   
                        . 94p.
                        , Social media has emerged as a tool for exploring the human psyche, offering exceptional
access to real-time behavioral signals that are transforming the landscape of computational
psychiatry. Among these, bipolar disorder is difficult to detect because of its volatility,
requiring robust modeling of emotion, behavior, and timing. Prior studies have largely
focused on text-based sentiment analysis and linguistic features to classify mental health
conditions; however, these approaches often neglect non-verbal markers such as circadian
rhythms and affective variability. Existing models primarily depend on static textual cues,
limiting their ability to capture the dynamic, multimodal nature of psychiatric expression.
This study addresses these limitations by integrating temporal rhythms, emotional
dynamics, and behavioral signals extracted from Reddit user histories to develop predictive
models of bipolar disorder and high-risk psychological states. We propose a series of
interpretable multimodal architectures employing classical machine learning (Logistic
regression, Random Forest, and XGBoost), deep sequence models (LSTM, GRU), and
transformer-based frameworks (Roberta, GPT). Our approach incorporates temporal
posting features, emotional entropy, and community-level interaction structures.
Compared to benchmark studies, our models demonstrate significant improvements in both
classification (F1 &gt; 0.99) and regression (R² &gt; 0.89), highlighting the predictive power of
fused behavioral signals. This work advances the field by providing a scalable, languageindependent framework for the early detection of psychiatric risk. It also holds broader
implications for public health by offering a foundation for real-time, ethically deployable
digital mental health tools.
                         30cm. 
                        
       </p>

<p><a href="http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-reserve.pl?biblionumber=615943">Place Hold on <em>Social Media Analytics for Mental Health Assessment /</em></a></p>

						]]></description>
       <guid>http://catalogue.nust.edu.pk:8081/cgi-bin/koha/opac-detail.pl?biblionumber=615943</guid>
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