NUST Institutions Library Catalogue Search for 'kw,wrdl: (su-br:"Synchronization.")'

Advances in 3G enhanced technologies for wireless communications

Boston, MA : Artech House, 2002 . xv, 350 p. : , Based on selected papers from the Fourth IEEE Workshop on Emerging Technologies in Circuits and Systems--Third Generation (3G) Mobile Technologies and Applications, held Nov. 29-Dec. 1, 2000, in Hong Kong. 24 cm.. 1580533027

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Software simulation of discrete multitone modem /

By Khan , NC Hassan Talha (TCC-11). Rawalpindi MCS (NUST) 2003 . 144 p.;

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Recent Advances in Biomedical Engineering (E-Book)

By Dr Ganesh R Naik. India In-Teh 2009 . xii,660 P;

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Mathematical Control Theory : An Interoduction/

. 260p. 9780817647322

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Identifying Neurophysiological Correlates of Frontotemporal Dementia: Resting State EEG and Phase Synchronization Analysis /

By Ali, Salwa . . 123p. , The need to develop more efficient neuropsychological biomarkers is paramount in the identification of neurodegenerative diseases, tracking the efficiency of treatment and in an effort to avoid the huge financial cost required. While previous research utilizing neuroimaging techniques has pinpointed changes in functional connectivity (FC) as promising biomarkers for frontotemporal dementia (FTD), the constraints of cost and availability of neuroimaging equipment underscore the necessity for accessible alternatives. Electroencephalography (EEG) has emerged as a viable option due to its increasing robustness, wider usage, and affordability. To this end, the research focuses on a resting-state EEG data created from AD, FTD, and HC groups. Here ground data were obtained from nineteen leads using a clinical EEG device when the subjects were in a resting state and their eyes were closed. Another challenge was to follow strict standards for data quality and quality management for data quality to enhance consistency. It is a cross-sectional study, including data from MiniMental State Examination conducted on each participant, and tapes recorded from 20 AD patients, 20 FTD patients, and 20 HC. The Neuroimaging Data Structure (BIDS) format was utilized to present both preprocessed and raw EEG data. The foremost aim was to determine the Feasibility, Sensitivity, and Specificity of the preprocessed, feature extracted, time-efficient, and artifact reduced EEG-derived FC patterns as markers in FTD. Phase-lock values (PLVs) were computed among nineteen pairs of electrodes across five frequency bands using MATLAB and the Hilbert transform. Significant variations in brain connectivity were identified through statistical analyses. The study revealed significant differences in alpha and beta frequency patterns among the control, Alzheimer's, and FTD groups, particularly in frontal and temporal regions. These differences suggest alterations in neural activity associated with cognitive processing, potentially serving as biomarkers for distinguishing between the three groups. Alterations in beta frequency PLV were noted across various EEG pairs, indicating disruptions in neural communication and coordination. These alterations suggest xvi compensatory mechanisms or hyperactivity in frontal and prefrontal regions, alongside potential cognitive and motor deficits due to decreased PLV in central and temporal regions. While no statistically significant differences were observed in delta and theta frequency synchronization between groups, trends suggest potential regions of interest for further research, aligning with existing literature exploring neural oscillations in neurodegenerative diseases. Similarly, no significant differences were observed in gamma frequency synchronization between groups, indicating relatively preserved neural synchronization in this frequency range across control, Alzheimer's, and FTD patients. In summary, both Alzheimer's and FTD demonstrate significant reductions in alpha and beta frequency values, particularly in frontal and temporal regions, compared to healthy controls. These findings underscore the altered functional network topology in AD and FTD, offering valuable insights into the neural mechanisms underlying these conditions. The study's results contribute to the development of electrophysiological markers, potentially enhancing the clinical diagnosis and understanding of AD and FTD. The specificity and sensitivity of EEG-derived FC patterns highlight their potential as costeffective, accessible biomarkers for neurodegenerative disease. 30cm.

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Therapeutic Potential of Light Flicker for Schizophrenia - A Comparative Study

By Ihsan ,Faryal. . 93p. ; , Schizophrenia is a serious neuropsychiatric condition that affects cognition, perception, and behavior. Current pharmacological treatments are successful in treating symptoms but can have serious adverse effects, especially in susceptible populations such as children and the elderly. These negative consequences, which range from metabolic problems to cognitive impairment, underline the critical need for alternate, non-invasive therapeutic techniques. One promising approach is neuromodulation by gamma entrainment with sensory stimulation, which uses an external stimulus to restore broken neuronal synchronization. This study looks at the combined effects of Clozapine and light flicker therapy in a ketamine-induced schizophrenia mouse model, with the hypothesis that their synergistic action will lead to improved recovery. Mice were classified into five groups: control, diseased, Clozapine-treated, light flicker-treated, and combination therapy. Behavioral examinations, such as the open field test, elevated plus maze, forced swim test, tail suspension test, novel object recognition, and Y-maze, demonstrated that combination therapy improved cognitive and affective deficiencies the most significantly. Histological analysis with hematoxylin and eosin staining of the cortex and hippocampus (CA1 and CA3) revealed severe neuronal disorganization in the diseased group, partial restoration in the individual treatment groups, and near-control level preservation in the combination therapy group. RT-PCR analysis using Pvalb and BDNF primers indicated abnormalities in PV interneurons and neuroplasticity markers, which corroborated the histological and behavioral findings. Our approach to schizophrenia combines neuroscience and psychology. Restoring gamma oscillations with light flicker improves PV interneuron function, BDNF production, and cognitive stability. This neuronal synchronization minimizes ego fragmentation, improves self perception, and promotes cognitive integration for a more comprehensive treatment. 30cm..

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Therapeutic Effects of Sound-Mediated Stimulation in a Mouse Model of Depression - A Comparative Study /

By Reemaz, Tooba . . 90p. , Depression is a prevalent neuropsychiatric disorder characterized by mood disturbances, cognitive impairment, and behavioral deficits. While pharmacological treatments such as Fluoxetine are widely used, they often represent limitations including delayed onset of action and side effects, highlighting the need for alternative, non-invasive therapeutic strategies. One promising approach is neuromodulation through gamma entrainment using auditory stimulation, aimed at restoring disrupted neuronal synchronization. This study evaluates the effects of 40 Hz sound stimulation compared to fluoxetine in a chronic restraint stress-induced mouse model of depression, hypothesizing that sound therapy will yield superior recovery outcomes. Mice were divided into control, depressed, fluoxetine-treated, and sound stimulation-treated groups. Behavioral assessments, including the Tail Suspension Test, Forced Swim Test, Social Interaction Test, Sucrose Preference Test, Open Field Test, and Light-Dark Box Test, revealed that 40 Hz sound therapy significantly alleviated depressive-like behaviors, often outperforming fluoxetine. Histopathological analysis focused on the hippocampus and prefrontal cortex showed that sound stimulation preventing neuronal loss and parvalbumin-expressing interneurons, which are crucial for inhibitory circuit function and gamma oscillatory activity. Molecular evaluation through qPCR for pavlb and brain-derived neurotrophic factor; BDNF supported these findings, displaying increased expression levels indicating enhanced interneuron integrity and neuroplasticity. This neuromodulatory treatment encourages the molecular and functional repair of brain circuitry implicated in depression. The findings demonstrated 40 Hz sound entrainment's ability to change brain oscillations and ameliorate cellular and behavioral deficiencies linked to depression, indicating that it may be a novel supplement or substitute for traditional antidepressant treatments. 30cm.

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